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One mechanism by which nettle root extract works is to block the binding of growth-stimulating estrogen to prostate cells (42-44, 48-50). When there is too little testosterone present, estrogen attaches to testosterone cell receptor sites throughout the body and creates many problems in aging men.
In youth, low amounts of estrogen are used to turn off the powerful cell-stimulating effects of testosterone. As estrogen levels increase with age, testosterone cell stimulation may be locked in the “off” position, thus reducing sexual arousal and sensation and causing the loss of libido so common in aging men (94, 99, 259). High serum levels of estrogen also trick the brain into thinking that enough testosterone is being produced, further slowing the natural production of testosterone.
This happens when estrogen saturates testosterone receptors in the hypothalamus region of the brain. The saturated hypothalamus then stops sending out a hormone to the pituitary gland to stimulate secretion of luteinizing hormone that the gonads require to produce testosterone.
High estrogen can thus shut down the normal testicular production of testosterone (1, 53, 54, 271-277). One further complication of excess estrogen is that it increases the body’s production of sex hormone-binding globulin (SHBG). SHBG binds free testosterone in the blood and makes it unavailable to cell receptor sites (51, 52, 55, 56). Based on the multiple deleterious effects of excess estrogen in men, aggressive action should be taken to reduce estrogen to a safe range if a blood test reveals elevated levels.
We will discuss the appropriate blood tests and steps that can be taken to lower estrogen levels later in this protocol. Testosterone is much more than a sex hormone. There are testosterone receptor sites in cells throughout the body, most notably in the brain and heart (60-180). Youthful protein synthesis for maintaining muscle mass and bone formation requires testosterone (67, 69, 81). Testosterone improves oxygen uptake throughout the body, helps control blood sugar, regulates cholesterol, and maintains immune surveillance (82, 83).
Testosterone is also a critical hormone in the maintenance of healthy bone density (59, 66, 67, 84-86), muscle mass, and red blood cell production (67, 69, 91-93, 98). Of critical concern to psychiatrists are studies showing that men with arimidex weight loss have lower levels of testosterone than do control subjects.
For some men, elevating free testosterone levels could prove to be an effective antidepressant therapy. There is a basis for free testosterone levels being measured in men with depression and for replacement therapy being initiated if free testosterone levels are low normal or below normal. Testosterone arimidex weight loss one of the most misunderstood hormones. Body builders tarnished the reputation of testosterone by putting large amounts of synthetic testosterone drugs into arimidex weight loss young bodies.
Synthetic testosterone abuse can produce detrimental effects (34), but this has nothing to arimidex weight gain with the arimidex weight loss a man over age 40 can enjoy by properly restoring arimidex weight loss natural testosterone to a youthful level.
Conventional doctors arimidex weight loss not recommended tes-tosterone replacement therapy because of an arimidex weight loss concern that testosterone causes prostate cancer. As we will later show, arimidex weight loss of arimidex on cycle cancer is not a scientifically valid reason to avoid testosterone modulation therapy. Another concern that skeptical physicians have about prescribing testosterone replacement therapy is that some poorly conducted studies showed it arimidex pct be ineffective in the long-term treatment of aging.
What physicians fail to appreciate is that exogenously administered testosterone can convert to estrogen in the body. The higher estrogen levels may negate the benefits of the exogenously administered testosterone. The solution to the estrogen-overload problem is to arimidex weight loss the conversion arimidex weight loss testosterone to estrogen in the liquid arimidex. A click at this page in Drugs and Aging (1999) click that androgen therapy can result in polycythemia (increased numbers of red blood cells) causing arimidex weight loss increase in blood viscosity and risk of clotting /side-effects-of-arimidex-for-breast-cancer/. For arimidex weight loss aging men, however, borderline anemia is a greater concern than red blood cell overproduction.
When men are arimidex prescribing information of arimidex weight loss during prostate cancer therapy, anemia frequently manifests.
Life Actually what is arimidex used for increases has not seen arimidex weight loss in which polycythemia developed in men taking enough tes-tosterone to restore physiological youthful ranges. In other words, too much testosterone could cause problems, but replacing arimidex weight loss to that arimidex weight loss a healthy 21-year-old arimidex weight loss not produce the side effects that some doctors are unduly concerned about.
As you will read in the section entitled “Testosterone and the Heart,” it appears that testosterone replacement therapy provides significant beneficial effects against cardiovascular disease.
Why Testosterone Levels Decline Testosterone production begins in the brain. When the hypothalamus detects a deficiency of testosterone in the blood, it secretes a hormone called gonadotrophin-releasing hormone to the pituitary gland. This prompts the pituitary to secrete luteinizing hormone (LH), which then prompts the Leydig cells in the testes to produce testosterone.
In some men, the testes lose their ability to produce testosterone, no matter how much LH is being produced. This type of testosterone deficiency is diagnosed when blood tests show high levels of LH and low levels of testosterone. So the pituitary gland vainly continues to secrete LH because there is not enough tes-tosterone in the blood to provide a feedback mechanism that would tell the pituitary to shut down.
In other cases, the hypothalamus, or pituitary gland, fails to produce sufficient amounts of LH, thus preventing healthy testes from secreting testosterone. Blood testing can determine whether sufficient amounts of LH are being secreted by the pituitary gland and help determine the appropriate therapeutic approach. If serum (blood) testosterone levels are very low, it is important to diagnose the cause, but no matter what the underlying problem, therapies exist today to safely restore testosterone to youthful levels in any man (who does not already have prostate cancer).
As indicated earlier, a major problem that aging men face is not low production of testosterone, but excessive conversion of testosterone to estrogen. Specific therapies to suppress excess estrogen and boost free testosterone back to youthful physiological levels will be discussed later. The Effects of Testosterone on Libido Sexual stimulation and erection begin in the brain when neuronal testosterone-receptor sites are prompted to ignite a cascade of biochemical events that involve testosterone-receptor sites in the nerves, blood vessels, and muscles.
Free testosterone promotes sexual desire and then facilitates performance, sensation, and the ultimate degree of fulfillment. Without adequate levels of free testosterone, the quality of a man’s sex life is adversely affected and the genitals atrophy.